💡 Spinal fractures range from low-energy osteoporotic wedge fractures to high-energy fracture–dislocations with cord injury. Your priorities are: ATLS, spinal precautions, early imaging, and neurology. Stability and neurological status drive management.
📖 Initial Priorities (ATLS + Spinal Precautions)
- ATLS first: Airway with in-line stabilisation, Breathing, Circulation. Avoid neck manipulation until cleared.
- Spinal precautions: Rigid collar (if not contraindicated), head blocks, scoop/slider, log-roll with team lead; avoid prolonged hard-board time (pressure sores).
- Analgesia & VTE: Early multimodal analgesia; LMWH when safe (balance with neuraxial procedures / haematoma risk).
- Safeguarding red flags: In the elderly, consider fragility fracture; in non-ambulant or inconsistent histories, consider non-accidental injury.
🧠 Neurological Assessment (drives urgency)
- Screen: Pain site, paraesthesia, power, sphincter tone, priapism (spinal shock), saddle anaesthesia.
- Classify: Use ASIA Impairment Scale (A–E); document dermatomes/myotomes (C5–T1, L2–S1) and perianal sensation/voluntary anal contraction.
- Red flags — act now: New deficit, suspected cauda equina (bilateral sciatica, urinary retention/incontinence, saddle anaesthesia), progressive weakness → urgent MRI and spinal/ortho-neuro referral.
🩻 Imaging Strategy
- CT whole spine is the workhorse in trauma — high sensitivity for bony injury; include reformats.
- MRI if neurological deficit, suspected ligamentous/cord injury, epidural haematoma, occult fracture, or for cauda equina; also in persistent pain with normal CT.
- Plain films only for low-risk scenarios (e.g. outpatient osteoporotic pain) where CT is not needed.
🧩 Injury Patterns — What they mean
- Cervical:
– C1 Jefferson (axial load; burst of atlas) — assess transverse ligament on MRI.
– C2 Odontoid (Type I tip; II base — unstable; III into body).
– Hangman’s (C2 pars) — hyperextension/axial load.
– Facet dislocations (unilateral/bilateral) — often with cord risk.
- Thoracolumbar (T/L):
– Compression/wedge (anterior column) — often stable, esp. osteoporotic.
– Burst (retropulsed fragments) — canal compromise, variable stability.
– Chance (flexion–distraction seat-belt injury) — posterior ligamentous complex (PLC) injury → unstable.
– Fracture–dislocation — multidirectional instability, high cord risk.
🧮 Classifying Stability (helps decisions)
- Denis 3-column model: Middle column injury ⇒ likely unstable.
- AO Spine (simplified):
– A (compression), B (tension band failure/PLC), C (translation/rotation).
Higher letter = higher instability; add neurological (N0–N4) and modifiers (M).
- C-spine clearing: Use validated rules (e.g., Canadian C-Spine) in low-risk; in the ED, most trauma goes straight to CT.
💊 Acute Medical Management (what you can start now)
- Pain: Paracetamol + short NSAID course if renal/GI risk acceptable; early short-course opioids; calcitonin can help acute osteoporotic pain (short-term).
- Immobilisation: Collar/halo for selected C-spine; TLSO brace for selected T/L stable patterns. Avoid over-immobilising frail patients (delirium, sores).
- Neurogenic shock: Hypotension + bradycardia (above T6). Treat with cautious fluids, vasopressors (e.g., noradrenaline). Maintain MAP targets (often ≥85–90 mmHg early) if cord injury suspected (local protocol).
- Steroids: Routine high-dose steroids are not recommended for acute cord injury in UK practice.
- Cauda equina syndrome: Emergency MRI and decompression — time-critical.
🔧 Surgical vs Conservative — The principles
- Operate for: unstable patterns (AO B/C), progressive or significant neuro deficit, significant canal compromise (burst with retropulsion + deficit), failure of conservative care, polytrauma requiring early mobilisation.
- Conservative for: stable compression fractures without deficit; good pain control; brace + physio + early mobilisation.
- Cement augmentation (vertebroplasty/kyphoplasty): selected painful osteoporotic compression fractures refractory to optimal medical therapy (exclude posterior wall breach/neural compromise).
🧱 Osteoporotic (Fragility) Vertebral Fractures
- Presentation: Sudden thoracolumbar pain after minimal trauma, height loss, kyphosis; neurology usually intact.
- Assess secondary causes: Ca²⁺, PO₄, ALP, 25-OH-vit D, TFTs, coeliac screen, SPEP/UPEP (myeloma), steroids history.
- Management: Analgesia, short rest then mobilisation, spinal orthosis if helpful, falls assessment, DEXA. Start bone protection per FRAX/DEXA (oral bisphosphonate first-line; IV zoledronate if adherence/GI issues); ensure calcium/vit D repletion.
🧪 Associated Syndromes to Spot
- Diffuse idiopathic skeletal hyperostosis / ankylosing spondylitis: “Long-bone” spine — fractures are highly unstable after minor trauma; require CT/MRI and specialist fixation strategies.
- Pathological fractures: Metastases, myeloma, infection — look for lytic lesions, constitutional symptoms; consider MRI and oncology/ID input.
🧰 Practical Ward Checklist
- ATLS done; C-spine protected; analgesia charted; bowel regimen started; LMWH considered.
- CT whole spine reported; MRI requested if neuro/PLC concern.
- ASIA grade documented; neuro obs frequency set; MAP goals communicated if cord injury.
- Bracing instruction written; PT/OT referrals; pressure-area care; delirium prevention.
- Bone health: FRAX/DEXA plan, Ca/vit D, bisphosphonate where indicated; falls clinic referral.
⚠️ Red Flags — Don’t Miss
- New neuro deficit; sphincter dysfunction; saddle anaesthesia → urgent MRI/consult.
- High-risk mechanisms (fall from height, high-speed RTC), midline tenderness, intoxication, distracting injuries → image liberally.
- Frail patient on the trolley for hours with collar → pressure sores and delirium: expedite clearance or appropriate padding.
📚 Teaching Pearls (why it works)
- Stability + Neurology are the two axes that predict deterioration and dictate fixation vs bracing.
- Middle column injury (Denis) or PLC failure (AO B) usually means instability even if pain seems modest.
- Early mobilisation (with safe bracing) reduces complications: VTE, pneumonia, deconditioning — particularly in older adults.
- Don’t chase x-ray “heights” alone: correlate pain, PLC signs, canal compromise, and the patient’s goals.
🧑⚕️ Case 1 — Cervical Trauma (High-energy)
A 32-year-old man is brought in after a high-speed RTC. He has neck pain, is unable to move his legs, and has urinary retention. CT shows a C5–C6 fracture-dislocation with cord compression.
Teaching point: This is an unstable fracture with neurological deficit. Priorities: ATLS, spinal precautions, urgent MRI, and referral for surgical decompression/stabilisation. Maintain MAP >85 mmHg to optimise cord perfusion. Document ASIA grade on admission.
🧑⚕️ Case 2 — Osteoporotic Compression
A 78-year-old woman develops sudden mid-back pain after bending to pick up shopping. CT thoracolumbar spine shows a stable L1 wedge compression fracture with no retropulsion. Neurology is intact.
Teaching point: This is a stable fragility fracture. Managed conservatively with analgesia, mobilisation, spinal orthosis (if needed), and secondary prevention (DEXA, bisphosphonates, vitamin D/calcium, falls assessment). No brace needed if pain is controlled.
🧑⚕️ Case 3 — Chance (Seat-belt) Injury
A 42-year-old man, restrained driver, has severe back pain after a road traffic collision. CT shows a horizontal fracture through T12 with posterior ligamentous complex injury. No neurological deficit.
Teaching point: A flexion–distraction (Chance) fracture is unstable. Even without neuro deficit, he requires spinal surgical input, often for stabilisation. Always consider associated intra-abdominal injuries (common with lap belt trauma).
🧑⚕️ Case 4 — Pathological Collapse
A 65-year-old man with known myeloma presents with worsening lumbar pain, night sweats, and weight loss. CT shows lytic destruction and collapse of L3 vertebral body with mild canal compromise. No motor deficit.
Teaching point: This is a pathological vertebral fracture. Management: analgesia, bracing, oncology referral, systemic therapy for myeloma, and consider vertebroplasty/kyphoplasty for pain control. Always investigate secondary causes (SPEP/UPEP, Ca²⁺, renal function).