π« Accelerated Idioventricular Rhythm (AIVR) is a transient ventricular rhythm often seen post-MI, particularly during reperfusion. It is usually benign, self-limiting, and haemodynamically well tolerated, so treatment is rarely required. The key challenge is distinguishing it from slow VT or CHB.
π About
- Commonly observed after reperfusion in acute MI (PCI or thrombolysis).
- Often resembles a "slow VT" but is not typically malignant.
- Most cases resolve spontaneously as sinus rate accelerates past the ventricular focus.
- Generally requires only observation and reassurance unless instability develops.
π ECG Characteristics
- Broad-complex rhythm, usually regular.
- Rate: 40β120 bpm (classic: 60β100). π
- AV dissociation often present (cannon A waves may be seen clinically).
- At rates >110β120 bpm, consider slow VT instead.
π§Ύ Differential Diagnosis
- CHB with escape rhythm: if rate <50 bpm.
- Slow VT: if rate >110β120 bpm, particularly in scarred ventricles.
- Junctional rhythm with aberrancy: consider if QRS morphology varies.
β οΈ Causes / Associations
- π Acute STEMI, especially reperfusion arrhythmia.
- Ischaemic heart disease, cardiomyopathy.
- Myocarditis, digoxin toxicity (check KβΊ and drug levels).
- Cocaine use.
- Normal finding in well-trained athletes (high vagal tone).
- During ROSC after cardiac arrest.
π©Ί Clinical Features
- Often asymptomatic, may have variable heart sounds due to AV dissociation.
- Seen in acute MI context β always check for associated complications.
- Usually haemodynamically stable (key teaching pearl: instability is unusual).
π Management
- Observation only in most cases β treat the underlying condition (e.g., MI).
- Spontaneously resolves once sinus node regains dominance.
- Do NOT suppress with antiarrhythmics (lidocaine, amiodarone) unless unstable β risk of asystole β οΈ.
- Seek senior input if in doubt, particularly in the peri-MI setting.
π References
Cases β Accelerated Idioventricular Rhythm (AIVR)
- Case 1 β Post-Reperfusion MI:
A 58-year-old man is admitted with an acute anterior STEMI. He undergoes primary PCI with stenting of the LAD. An hour later, telemetry shows a wide-complex rhythm at 70 bpm with gradual onset and offset. He is asymptomatic, BP stable, and the rhythm spontaneously reverts to sinus.
Diagnosis: AIVR following reperfusion therapy for MI.
Management: No treatment needed β benign rhythm. Monitor and continue post-MI care (dual antiplatelets, beta-blocker, ACEi, statin).
- Case 2 β Digoxin Toxicity:
A 72-year-old woman with atrial fibrillation on digoxin presents with nausea and visual halos. ECG: regular wide-complex rhythm at 60 bpm, independent of atrial activity. Serum digoxin level high.
Diagnosis: AIVR associated with digoxin toxicity.
Management: Stop digoxin, correct electrolytes (especially potassium and magnesium), consider digoxin-specific antibody fragments if severe. Continuous cardiac monitoring until rhythm resolves.
Teaching Commentary β‘
AIVR is a ventricular rhythm faster than the intrinsic ventricular escape rate (β40 bpm) but slower than VT (usually 50β110 bpm). It typically occurs after reperfusion of acute MI (a βreperfusion arrhythmiaβ), but also in drug toxicity (digoxin), cardiomyopathy, and metabolic disturbances. ECG shows a regular, wide-complex rhythm with gradual onset/offset.
Unlike VT, AIVR is benign and self-limiting, rarely requiring treatment. Management is to treat the underlying cause (e.g. reperfusion, stop digoxin, correct electrolytes) rather than antiarrhythmics.